Recent epidemiological and clinical research on human
adipocytes now tells us that intakes of high-Calcium
may reduce Calcium concentrations in fat cells by
lowering the production of two hormones – Parathyroid
Hormone (PTH) and a hormonal, active form of Vitamin
D. This in turn, allows break down and release of
fat and suppresses the formation of new fat.
Readily water soluble calcium salts are easily
absorbable like the Calcium as in CAL-SAP™
plays an important role in preventing obesity, by
controlling or rather lowering energy metabolism.
You need not worry at all, if you have over-eaten
a high-calorie diet, Calcium in CAL-SAP™ is
there to regulate your weight gain even during this
period.
An important fact on the role of Calcium against
our body weight may be considered in this
connection –
Studies from observations indicate that there is
an inverse relationship between our body weight
and the combined intakes of dietary Calcium and
normal protein.
Observations from a randomized, crossover study
of 3 isocaloric 1-week diet reveal the following
–
|
Low
Calcium and Normal Protein
LC / NP |
500
mg. Calcium
15% of energy from protein |
|
High Calcium and Normal
Protein
HC / NP |
1800 mg. Calcium
15% of energy from protein |
|
High
Calcium and High Protein
HC / HP |
1800 mg.
Calcium
23% of energy from protein |
An interesting result emerged from this observational
study of HC / NP diet.
The fecal fat excretion increased by ~ 2.5 –
fold.
| HC
/ NP |
14.2 g.
/ day |
| LC / NP
|
6.0 g. / day |
| HC
/ HP |
5.9 g. / day |
Not only this, in case of HC / NP diet, the fecal
energy excretion increased by ~ 86.6 Kcal / day,
compared with LC/NP and HC / HP diets.
These observational studies henceforth explain
why a combined intake of dietary Calcium and normal
protein may oppose the accretion of excess body
weight.
Sources:
1 Office of Dietary Supplements, NIH Clinical Centre,
National Institutes of Health
http://ods.od.nih.gov/factsheets/calcium.asp
2 Regulations of Adiposity and Obesity Risk by
Dietary Calcium: Mechanism and Implications, Michael
B. Zemel, Ph.D., The University of Tennessee, Knoxville,
Tennessee.
3 Effect of short – term high calcium intake
on 24 – h energy expenditure, fat oxidation
and fecal fat excretion, International Journal of
Obesity (2005)29, 292 – 301.
